Excessive calcium ingestion leading to milk-alkali syndrome

Ann Clin Biochem 2008;45:527-529
doi:10.1258/acb.2008.008006
© 2008 Association for Clinical Biochemistry

 

 

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Case Reports


C S Bailey1,
J J Weiner2,
O M Gibby3 and
M D Penney1


1 Department of Clinical Biochemistry;
2 Department of Obstetrics and Gynaecology;
3 Department of Endocrinology, Royal Gwent Hospital, Cardiff Road, Newport, South Wales NP20 2UB, UK


Corresponding author: Catherine S Bailey. Email: Catherine.Bailey{at}gwent.wales.nhs.uk

This report describes the presentation and clinical course of a 40-year-old woman who had an emergency admission for eclampsia. During routine investigations, she was found to have profound hypercalcaemia, the cause of which was identified as milk-alkali syndrome, caused by self-medication with antacid tablets for dyspepsia. Treatment with aggressive rehydration, bisphosphonates and discontinuation of antacid tablets restored normocalcaemia. The patient made a full recovery with no long-term side-effects. Her male infant was safely delivered with no deleterious effects of exposure to high calcium concentrations in utero.


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